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3924 18th Ave NE, Olympia WA 98506 MLS #695338
Kinga Mills - Coldwell Banker Danforth Search 1000 #39;s of properties at http://www.buysellwahome.com Beautifully updated home with an amazing Yard, RV parking, sprinkler system, and a Hot Tub!...
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3924 18th Ave NE, Olympia WA 98506 MLS #695338 - Video
is there anyone out there - Two Sheds Jackson
from the album "memory lane"
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is there anyone out there - Two Sheds Jackson - Video
Nightcore: Jasonayeiter-- Joe DeRosa Sheds Emo Tears
I actually like Joe DeRosa and generally love it when he shows up on Opie and Anthony. However, there have been some problems between him and Anthony recently and Jasonayeiter, one of the most...
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Nightcore: Jasonayeiter-- Joe DeRosa Sheds Emo Tears - Video
Enormous Whitetail Sheds. I #39;m an avid shed hunter
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Enormous Whitetail Sheds. I'm an avid shed hunter - Video
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Set in a lawless town in the Austrian mountains, the English-language "Autumn Blood" seems at first like something along the lines of "The Sound of Music" as re-imagined by Michael Haneke. But whereas Haneke's films grapple with the blunt force of violence, novice filmmaker Markus Blunder just lets the violence snowball all the way down a slippery slope.
Nameless siblings (Sophie Lowe and Maximilian Harnisch) witness the senseless murder of their father. They lose their mother a few years later. Ostracized by the small foothill community for some unknown reason, the orphans carry on with their routines without telling anyone. Three local thugs (Samuel Vauramo, Gustaf Skarsgard and Tim Morten Uhlenbrock) seize every opportunity to molest and assault the teenage girl, sometimes right in front of her preteen brother.
One might wonder if someone in this situation would commence a self-defense regimen or at the very least reinforce the door locks. So it's frustrating to watch this character run and hide (poorly) with her dress stripped off for no apparent reason. The gratuitous nudity raises suspicions of whether Blunder is on some level blaming the victim.
"Autumn Blood."
MPAA rating: R for violence including rape, nudity.
Running time: 1 hour, 40 minutes.
Playing: At AMC Universal CityWalk Stadium 19. Also on VOD.
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'Autumn Blood' sheds gratuitous violence
PUBLIC RELEASE DATE:
18-Sep-2014
Contact: Phil Sahm phil.sahm@hsc.utah.edu 801-581-2517 University of Utah Health Sciences
(SALT LAKE CITY)A new study by researchers at the University of Utah School of Medicine sheds light on a longstanding question about the role of mitochondria in debilitating and fatal motor neuron diseases and resulted in a new mouse model to study such illnesses.
Researchers led by Janet Shaw, Ph.D., professor of biochemistry, found that when healthy, functioning mitochondria was prevented from moving along axons nerve fibers that conduct electricity away from neurons mice developed symptoms of neurodegenerative diseases. In a study in the Proceedings of the National Academy of Sciences, Shaw and her research colleagues said their findings indicate that motor neuron diseases might result from poor distribution of mitochondria along the spinal cord and axons. First author Tammy T. Nguyen, is a student in the U medical school's M.D./Ph.D. program, which aims to produce physicians with outstanding clinical skills and rigorous scientific training to bridge the worlds of clinical medicine and basic research to improve health care.
"We've known for a long time of the link between mitochondrial function and distribution and neural disease," Shaw says. "But we haven't been able to tell if the defect occurs because mitochondria aren't getting to the right place or because they're not functioning correctly."
Mitochondria are organelles compartments contained inside cells that serve several functions, including making ATP, a nucleotide that cells convert into chemical energy to stay alive. For this reason mitochondria often are called "cellular power plants." They also play a critical role in preventing too much calcium from building up in cells, which can cause apoptosis, or cell death.
For mitochondria to perform its functions, it must be distributed to cells throughout the body, which is accomplished with the help of small protein "motors" that transport the organelles along axons. For the motors to transport mitochondria, enzymes known as Mitochondrial Rho (Miro1) GTPases act to attach mitochondria to the motors. To study how the movement of mitochondria is related to motor neuron disease, Nguyen developed two mouse models in which the gene that makes Miro1 was knocked out. In one model, mice lacked Miro1 during the embryonic stage. A second model lacked the enzyme in the cerebral cortex, spinal cord and hippocampus.
The researchers observed that mice lacking Miro1 during the embryonic stage had motor neuron defects that prevented them from taking a single breath once born. After examining the mice, Nguyen, Shaw and their colleagues discovered that neurons required for breathing after birth were missing from the upper half of the mice's brain stems. The phrenic nerve, also important for breathing, was not fully developed, either.
"We believe the physical difficulties in the mice indicated there were motor neuron defects," Shaw says.
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Mouse model sheds light on role of mitochondria in neurodegenerative diseases
Depictions of animals in ancient Egyptian artefacts have helped scientists assemble a detailed record of the large mammals that lived in the Nile Valley over the past 6,000 years. A new analysis of this record shows that species extinctions, probably caused by a drying climate and growing human population in the region, have made the ecosystem progressively less stable.
The study, published in Proceedings of the National Academy of Sciences (PNAS), found that local extinctions of mammal species led to a steady decline in the stability of the animal communities in the Nile Valley. When there were many species in the community, the loss of any one species had relatively little impact on the functioning of the ecosystem, whereas it is now much more sensitive to perturbations, according to first author Justin Yeakel, who worked on the study as a graduate student at the University of California, Santa Cruz, and is now a postdoctoral fellow at the Santa Fe Institute.
Around six millennia ago, there were 37 species of large-bodied mammals in Egypt, but only eight species remain today. Among the species recorded in artwork from the late Predynastic Period (before 3,100 BC) but no longer found in Egypt are lions, wild dogs, elephants, oryx, hartebeest, and giraffe.
"What was once a rich and diverse mammalian community is very different now," Yeakel said. "As the number of species declined, one of the primary things that was lost was the ecological redundancy of the system. There were multiple species of gazelles and other small herbivores, which are important because so many different predators prey on them. When there are fewer of those small herbivores, the loss of any one species has a much greater effect on the stability of the system and can lead to additional extinctions."
Egyptian statues image via Shutterstock.
Read more at Bristol University.
Originally posted here:
Egyptian art sheds light on changing ecosystem
Sony acquired game streaming company Gaikai back in 2012, and today Game Informer published a wide-ranging interview with Gaikai CEO David Perry that (among other things) sheds some light on how that deal came about.
"We were very worried because we were doing only PC games," says Perry, who goes on to run down the list of troubles Gaikai had while trying to get PC builds of games streaming through their network: translating keyboard and mouse control layouts to TV-friendly gamepads, translating resolutions in real-time and sorting out credentials for proprietary publisher platforms like Games For Windows Live.
"We wanted to get [on a platform] where there was a large set of very high-quality games but with a standardized control system, something that used a joypad that all the games would be compatible with," says Perry. "And it didnt have crazy random security systems included."
Perry also said with PlayStation Now being built into Sony TVs, the service will be able to offer higher-quality games than what mainstream TV users are used to. "[You] go to the games section [of your new TV] and youre like 'Oh man, its bowling and darts.' [But] thats where we need to put The Last of Us and the best that our industry can make."
Sony (and, by extension, Perry) has previously said that Gaikai's technology suite will one day be capable of streaming old games -- think PlayStation 1, 2 and 3 -- on contemporary consoles. The current version of PlayStation Now is capable of streaming PlayStation 3 titles, but Perry says that going back farther requires further work on the Gaikai technology platform and support directly from Sony's Japanese division, since it's not like he can easily ask the individual developers on those games to dig back into their source code and build in support for PlayStation Now.
"You want to always try to think about how you can unlock the past. I would love to be able to unlock every game ever on any 3D platform," says Perry. "I cant do that by requiring all the [old] teams to touch their code...that would be Sony of Japan doing this."
The full interview, which contains more technical details about Gaikai's operating structure and the challenges it faces as global broadband speeds slowly grow, can be read over on the Game Informer website.
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New interview sheds light on PlayStation Now's past, future
